Clinical

Alzheimer's Disease

The most common neurodegenerative disease, characterized by amyloid-β plaques, tau tangles, neuroinflammation, and progressive cognitive decline.

In Depth

The ECS is significantly dysregulated in Alzheimer's disease — CB1 receptor density is reduced in affected brain regions, while CB2 receptors are upregulated on activated microglia surrounding amyloid plaques. Preclinical studies show cannabinoids reduce amyloid-β production, tau hyperphosphorylation, and neuroinflammation. THC has been shown to directly inhibit acetylcholinesterase and prevent amyloid aggregation in vitro. Small clinical studies suggest cannabis may reduce agitation and improve sleep in Alzheimer's patients, but large RCTs are lacking.

Further Reading